Cure Myasthenia Gravis

Autoimmune Paleo Cookbook

If you have an autoimmune disease, recipes can often be hard to find and you are often told the huge amounts of things like chocolate and certain foods with too high of a fat content that you can and can't eat. This eBook gives recipes that anyone can prepare without too much trouble. Even if you don't like cooking, this book makes cooking easy and breaks it down into steps. Best of all, the recipes do not taste like healthy medicine recipes. These recipes are delicious foods that anyone would want to eat, even if they didn't have to eat healthy. This book contains over 70 amazing recipes for anyone with an autoimmune disorder. The book comes with two free ebooks: 7 Steps to Living Well With an Autoimmune Disorder and The Top 10 Autoimmune Diseases Checklist. If you want to learn about your autoimmune disease and the best and worst foods for you, this is the book for you!

Autoimmune Paleo Cookbook Summary


4.6 stars out of 11 votes

Contents: Ebook
Author: Samantha Miller
Price: $27.00

My Autoimmune Paleo Cookbook Review

Highly Recommended

The author has done a thorough research even about the obscure and minor details related to the subject area. And also facts weren’t just dumped, but presented in an interesting manner.

Purchasing this ebook was one of the best decisions I have made, since it is worth every penny I invested on it. I highly recommend this to everyone out there.

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Controlling Toxic Risk

Wellness is the state of existence that arises when health-sustaining homeostatic balance is gained and maintained. Individual and cumulative toxic exposures threaten this optimal homeostatic state. However, identifying and compensating for toxic exposure can minimize the detrimental effects of the exposure. General signs, symptoms, and risks that may indicate the presence of a high risk for toxic load include diabetes congestive heart failure obesity history of alcohol abuse psoriasis and other skin disorders heavy exposure to industrial or household chemicals frequent or recurrent use of medications use of hormonal therapy, including hormone replacements and oral contraceptives and disease states that alter liver, kidney, or GI functioning. Also, due to the sensitivity of the immune system, immunologic disorders may also be seen with toxic exposures such as autoimmune diseases, immune suppression, and chronic inflammation. The review below of the mechanisms of some of the most...

Conventional Therapies

Multiple sclerosis or experimental autoimmune encephalomyelitis (EAE), circulating lymphocytes and monocytes macrophages readily cross the blood-brain barrier and gain access to the CNS leading to edema, inflammation, and demyelination. Also often used to modify the disease process in MS is glatiramer acetate, a mixture of synthetic polypeptides composed of four amino acids, and based on the structure of MBP, which is believed to inhibit the T-cell response to multiple antigens in myelin. Glatiramer acetate induces T-regulatory cells known as GA-specific regulatory CD4 and CD8 lymphocytes, as well as causing a shift from Th1 to Th2 activity, increasing the secretion of anti-inflammatory cytokines and suppressing the autoimmunity led by Th1 cells.18

Quality issues with echinacea products

As outlined at the beginning of this chapter, monographs on Echinacea and anecdotal accounts often refer to limitations on its use. In particular, there is referral to the concept that Echinacea will cause a tachyphylaxis in immune response and hence should only be used for 5 days or so. Certainly continuous use is not advised by several sources. Also it is often written that Echinacea is contraindicated in autoimmune disease. The origin of this highly cautious approach to what is a relatively benign agent needs to be critically examined. The extensive range of conditions for which the Eclectics prescribed Echinacea is summarized in Table 14.1. It is clear from this table that the limitations on Echinacea suggested by modern writers are not supported. The conditions in the table are mainly infections and envenomations of various kinds (which clearly attest to Echinacea's influence on the immune system). However, the inclusion of tuberculosis and disorders related to autoimmunity such...

Echinacea as an immunomodulator

That Echinacea works best as a preventative is consistent with the clinical experience of many phytotherapists, although this activity has not been borne out by recent studies (Melchart and Linde, 1999). In fact, it may be more accurate to describe Echinacea as an immunomodulator. While it stimulates phagocytic activity, this may have the end result of modulating immune function overall. For example, the chronic presence of a microorganism may cause a state of immune dysregulation that results in autoimmune disease or a chronic inflammatory condition such as asthma. Such theories have been proposed in the mainstream scientific literature (Bone, 1999). A substance that enhances immune surveillance may help the body to eliminate the organism or neutralize its imbalancing effect on the immune system, thereby toning down an inappropriate immune response. Similarly, the body's response to an allergen may be reduced if a more appropriate response results from enhanced phagocytic activity...

The Hygiene Hypothesis

During the first six months of life, and avoidance of frequent antibiotic administration) may restore the T-helper subset balance, resulting in fewer incidences later in life of asthma and allergy. It appears that these exposures must occur prior to the first year of life to make a difference. Experimental use of mycobacterial strains has demonstrated a shift from Th2-immune responses to Thl-immune responses, thereby preventing the allergy development in mice, as well as ameliorating autoimmune diseases characterized by Th1 responses.7 It is interesting to note that both autoimmune and allergic diseases share a parallel increasing prevalence. Rebalance of the Th1- and Th2-subset cell ratios is a highly speculative theory because it does not explain fully the complete immunologic etiology of asthma and allergies.

Etiology And Epidemiology

The etiology of MS is unknown, but evidence suggests that genetic, environmental, and immunologic factors and infectious agents may be involved. Currently, MS is presumed to be an autoimmune disease that develops in genetically susceptible individuals upon activation by some unknown environmental trigger. Several risk factors have been associated with MS. Tobacco smoking significantly increases the odds of developing MS.2 So do other lifestyle factors, such as drinking coffee and alcohol.3 Women are two to three times more likely to develop MS than men, and Caucasians, particularly those of Northern European descent, are more likely to develop MS than other ethnic groups. Individuals living at northern latitudes are also at increased risk, suggesting a possible link with vitamin D and sun exposure. In addition, migration from one geographic area to another changes an individual's risk for MS.

Etiology And Pathophysiology

Studies have found a greater frequency of certain traits and characteristics among parents of autistic than of healthy children. Greater maternal and paternal age are independently associated with an increased risk for ASD in offspring.7 Additionally, mothers who have allergies and asthma during the second trimester of pregnancy have a twofold greater than average risk of having a child with ASD. Although this same study found maternal psoriasis to be the only autoimmune disease associated with an increased risk for ASD,8 another study found a link between both maternal ulcerative colitis and paternal type 1 diabetes and increased risk for infantile autism.9 Some autistic children exhibit auto-antibodies to brain proteins such as myelin basic protein (anti-MBP) and neuron-axon filament protein (anti-NAFP). One study found a correlation between these auto-antibodies and antibodies directed against measles virus (measles-IgG) and human herpes virus-6 (HHV-6-IgG). This study found that...

Preface for the ESP series in Photochemical and Photobiological Sciences

The changed recreational behavior and the correlation with several diseases in which sunlight or artificial light sources play a major role in the causation of clinical conditions (e.g. porphyrias, polymorphic photodermatoses, Xeroderma pigmentosum and skin cancers) have been well documented. As a result, in some countries (e.g. Australia) public services inform people about the potential risk of extended periods of sun exposure every day. The problems are often aggravated by the phototoxic or photoallergy reactions produced by a variety of environmental pollutants, food additives or therapeutic and cosmetic drugs. On the other hand, if properly used, light-stimulated processes can induce important beneficial effects in biological systems, such as the elucidation of several aspects of cell structure and function. Novel developments are centered around photodiagnostic and phototherapeutic modalities for the treatment of cancer, artherosclerosis, several autoimmune diseases, neonatal...

TLR Specificity for PAMPs in the Ectodomain and Adapters in the TIR Domain Underlie a Dual Recognition Response System

Molecular mechanisms that control TLR signaling specificities are obscure at present and need to be elucidated further. Indeed, it is difficult to explain how putative ligand-recognition motifs within the leucine-rich, N-terminal domains enable surprisingly broad TLRs ligand specificities, especially evident for TLR2 and TLR4. In addition, no compelling evidence is available for direct agonist binding by various TLRs. However, TLR-mediated signaling can be blocked by antibodies raised against the N-terminal region of the respective TLRs (Flo et al. 2002 Uehori et al. 2003), and mutations in the extracellular region impair TLR signaling and are associated with infectious and autoimmune diseases (see below). Thus, it is plausible that TLRs respond to PAMPs presented by various accessory molecules primarily by engagement of their ectodomain. Vogel et al. (2003) proposed that interaction of different combinations of adapter molecules and kinases with individual TLRs yields distinct...

The Rda For Iodine

The suggested daily RDA of 150 mg per day for iodine may be influenced by the fear that an excess of iodine can cause diseases including hyperthyroidism, hypothyroidism, goiter, rashes, and iodine allergy. However, most of these reactions are caused by pharmacologic doses of iodine. Some research has linked iodine excess to autoimmune thyroid disease, which can cause both hypo- and hyperthyroidism,5,6 but such autoimmune disease has been increasing

Hormone Balancing

Research suggests that hormones influence the duration and severity of autoimmunity affecting the CNS. One study found abnormally low levels of testosterone in human males with MS, and animal models of MS have shown low levels of testosterone and increased levels of luteinizing hormone,54 as well as an inverse relationship between testosterone levels and levels of inflammatory mediators.54 Another study showed improvement in cognitive performance, a slowing of brain atrophy, and increased lean body mass upon supplementation with a gel containing 100 mg of testosterone given daily to men with relapsing-remitting MS for a 12-month period.55 However, the supplementation had no significant effect on the numbers or volumes of sclerotic lesions.55 Levels of the androgen dehydroepiandrosterone (DHEA) have also been found to be significantly lower in MS patients than in healthy individuals.56

Kerry Bone

Traditional Use Does Not Support Limitations Modern Research Does Not Support Limitations Echinacea in Autoimmunity and Asthma Echinacea as an Immunomodulator Conclusions References only be used as a short-term treatment. Stimulation of the immune system will be detrimental in autoimmune disorders (such as multiple sclerosis) or in disorders where a heightened immune response may be counterproductive (such as AIDS, asthma, leukemia, and tuberculosis) hence Echinacea is con-traindicated in their treatment (Blumenthal et al., 1998).