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/ proteins for new cell wall synthesis

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Figure 7.7 A speculative view of the immediate consequences of auxin binding to a plasmalemmal (A) or cytoplasmic (B) receptor. PIP2, phosphatidyl inositol 4,5-bisphosphate; ER, endoplasmic reticulum; R, receptor; IP3, inositol 1,4,5-triphosphate; DG, diacylglycerol (from Brummell and Hall, 1987).

(Figure 7.9). In this example, and with many other auxins tested, the maximum value of the rate of efflux was always similar, with the unexplained exception of benazolin. Large-scale differences in the activity observed were attributed to the relative affinity of each herbicide for the auxin receptor in this species, which were computed by reference to a model of hormone-receptor interaction. These H50 values (the auxin concentration giving half maximal response) enable a more accurate and relevant comparison of auxin activity than data from growth bioassays, and may offer a means for a more detailed understanding of the auxin receptor in monocotyledonous species in the presence of herbicides.

Figure 7.8 Approximate differences in electrical potential and pH in a plant cell.
Figure 7.9 Dose-response curves for auxin-induced proton-efflux (modified from Fitzsimons et al., 1988).

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