Lose Weight By Controlling The Fat Storage Hormone

Beta Switch Program

Beta Switch Program is a proven, science-based, enjoyable weight loss program aimed at fighting stubborn fats in women. It was designed to specifically switches on the fat-burning power of stubborn female trouble spots, without restricting your favorite foods or doing excessive exercises. This is a System that allows women to achieve and experience freedom from obsessive-compulsive dieting, damaging workouts and negative body image. The plan was not created to be a quick fix. In fact, like every weight loss programs, it is hard; yet the easiest that can be. The system requires their full attention, being constant, and their discipline. For the period of its usage, they will have the opportunity to eat their favorite food without much fear. The only difference this time is that they will be eating it strategically. The methods employed in this book are natural ones that have been tested scientifically. The system comes with various bonuses- The Beta Switch Workout System, the 5-Day Tummy Tuck, Boost Your Body Image Report, Mind Over Matter: Win the Mental Game of Fat Loss and 1 full month in the exclusive Tight n' Toned club. Read more here...

Beta Switch Program Summary

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Contents: Ebooks, Videos
Author: Sue Heintze
Official Website: www.tight-n-toned.com
Price: $52.00

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My Beta Switch Program Review

Highly Recommended

All of the information that the author discovered has been compiled into a downloadable ebook so that purchasers of Beta Switch Program can begin putting the methods it teaches to use as soon as possible.

All the testing and user reviews show that Beta Switch Program is definitely legit and highly recommended.

Trouble Spot Nutrition

Created by Janet Hradil, Trouble Spot Nutrition is a 3 Phase Hormonal Solution That Melts Away Trouble Spot Fat In Less Than 15 Minutes A Day. Leptin, cortisol, and testosterone all have an influence on our weight issues, but not many of us know it. Janet Hradil has created Trouble Spot Nutrition with the intent of teaching people how their hormones affect their weight loss efforts, and how nutrition can easily correct hormone issues and help fight fat faster than ever before. In each of your fat cells, there is an enzyme, 11 beta-hydroxysteroid dehydrogenase-1 (Hsd), that takes inactive cortisone (a hormone) and turns it into cortisol, a fat storing compound. If you have high amounts of Hsd, you will have high amounts of fat storage. While Hsd is genetically determined, you can use nutrition to reduce levels and stop the unwanted fat storage, even on your trouble spots. Read more here...

Trouble Spot Nutrition Summary

Contents: Ebook
Author: Janet Hradil
Official Website: www.troublespotnutrition.com
Price: $15.00

How The Adrenal Glands Respond To Stress

At the center of the stress response are the adrenal glands. The adrenals produce epinephrine and norepinephrine, along with other hormones such as cortisol that enable the body to adapt to and survive a stressor. The acute alarm, or immediate reaction to a stressor, is a physiologic phenomenon in which the sympathetic nervous system responds to exogenous or endogenous stressors that put the body into what is popularly called the fight, fright, or flight'' mode. When the body is in this state, such as in potentially life-threatening situations, adrenal hormones are released to increase heart rate and blood pressure and divert blood to the brain, heart, and skeletal muscles. This physiologic compensation is a key mechanism in stress and the clinical phenomenon of adrenal fatigue. The adrenal cortex produces steroid hormones, which include cortisol, corticosterone, testosterone, estrogen, 17-hydroxyketosteroids, dehydroepiandroster-one (DHEA), DHEA sulfate, pregnenolone, aldosterone,...

Biochemical Reactions To Stressors

Following normal diurnal patterns for the release of serum cortisol, glucocorticoid levels are at their lowest point at approximately midnight to 1 am. Peak levels occur between 6 am and 8 am. Research has shown that elevation or suppression of daily cortisol levels indicates imbalanced hypothalamus-pituitary-axis (HPA) activity.12 This may be interpreted as hyper-function or hypofunction, depending on a patient's levels of cortisol and his or her clinical presentation. Sustained activation of the sympathetic nervous system marks the compensation stage, also known as the adrenal hyperfunction stage, with a secondary influence on the HPA axis. The pituitary gland responds to the influence of sympathetic nervous system activity by releasing adrenocorticotropic hormone (ACTH). In turn, the adrenal glands respond to the pituitary release of ACTH by producing excess cortisol and androgen hormones. In an attempt to compensate for this faulty hyperfunction, the HPA becomes less sensitive to...

Nutraceuticals For Treating Adrenocortical Dysfunction

Regenerate methionine Orally, prescribed PS product has decreased plasma cortisol and ACTH levels in healthy research subjectsc A cofactor for most ATP-dependent reactions and activation of intracellular secondary messenger cAMP Zinc deficiency increases membrane susceptibility to oxygen free-radical damage aKodama M, Inoue F, Kodama T. Intraperitoneal administration of ascorbic acid delays the turnover of labeled cortisol in plasma of ODA rat, but not Wistar rat Evidence in support of the cardinal role of vitamin C in the progression of glucocorticoid synthesis. In Vivo 1996 10 97-110 psychological stressor, phosphatidylserine supplementation blunts serum cortisol, ACTH, and salivary cortisol levels.107 Additionally, research has shown that phosphatidylserine supplementation in individuals with above-average neuroticism scores reported better moods and feeling less stressed.108 Results also indicate that phosphatidylserine improves cognitive function on tasks such as learning and...

Life histories reproductive strategies and allocation

It has been suggested that, to some extent, plants can escape the seed size--number trade-off by modifying the chemical composition of their seeds (Lokesha et al., 1992). There is some evidence that light wind-dispersed seeds are better dispersed than heavier seeds of similar morphology (e.g. Meyer & Carlson, 2001), although this may not apply to all dispersal modes (Hughes et al., 1994a). Since fats yield about twice the energy of carbohydrates per unit mass, a plant could make seeds half as heavy by replacing stored carbohydrate with fats. In fact, the majority of plant species mostly store fats in their seeds, although there are costs lipid synthesis is more energetically demanding than either protein or carbohydrate production. If lighter seeds are dispersed more effectively by wind, then we might expect fat storage to be more prevalent in wind-dispersed seeds than in those dispersed by other means. An analysis of a large dataset by Lokesha et al. (1992) supported this...

Natural Supports For Gaining And Maintaining Muscle Mass

Hormones have also been shown to have a great impact on insulin resistance. Stress and its resulting increase in epinephrine and cortisol affects insulin resistance. These adrenal hormones have been shown to increase glycogen breakdown from the liver and affect glucose utilization unfavorably.5,6 Estrogen supplementation may also increase insulin resistance, particularly in postmenopausal women.7 Increased testosterone in females and increased estrogens in males decrease peripheral glucose utilization.8 Studies have shown that dehy-droepiandrosterone (DHEA) supplementation increases peripheral glucose utilization.9 Insulin-like growth factor-1 (IGF-1) is a polypeptide stimulated by growth hormone, which affects growth and glucose metabolism. Studies have shown that IGF-1 increases peripheral glucose utilization as well as decreases protein catabolism.10

Dehydroepiandrosterone

Dehydroepiandrosterone (DHEA) is synthesized in the adrenal glands, liver, testes, and brain. This substance is converted to androstenedione, which is the precursor to other androgens, and improves insulin sensitivity. DHEA levels begin declining at approximately age 25. Studies have indicated that DHEA supplementation improves insulin sensitivity and decreases both subcutaneous and visceral fat.56 Specifically, animal studies have shown that DHEA decreases both body weight and the cytokine TNF-alpha, which is implicated in causing insulin resistance.57 A six-month study on adults showed that DHEA supplementation at 100 mg per day increased IGF-1. However, only the male subjects had decreased fat-body mass and increased muscle strength. Women had increases in total-body mass and had androgen levels that were above normal. No changes were seen in cortisol levels, lipid profiles, glucose levels, fasting insulin levels, bone-mineral density, or basal metabolic rates.58

Targeting to the Recycling Endosome

Rab11A, and its close relatives Rab11B and Rab25, have been shown to regulate recycling pathways for a number of cellular processes in animal cells, including polarized epithelial transport, and the insulin-dependent delivery of the GLUT4 glucose-transporter to the plasma membranes of fat cells (Cox et al. 2000 Wang et al. 2000 Wilcke et al. 2000). In addition, in these cells many of the proteins that cycle between the plasma membrane and internal compartments accumulate in the recycling endosome due to the slow rates of sorting and exit from these compartments (Maxfield and McGraw 2004). Even in animal systems, precise identification of sorting determinants and characterization of vesicle transport intermediates leading to and from these compartments remains poorly understood. In part, the lack of understanding

Strategies for Preventing Metabolic Syndrome

However, one does not need to look far for the basic causes of metabolic syndrome. All four conditions (obesity, hypertension, hypertriglyceridemia, and hyperinsulinemia) can be linked to one related cause poor dietary choices, namely, imbalanced consumption of simple carbohydrates. Thus, the main treatment for metabolic syndrome is dietary therapy. One study in women with a family history of cardiovascular disease, following a low-GI diet for four weeks, resulted in increased insulin sensitivity after a glucose challenge and increased glucose uptake in isolated fat cells. Even in lean young adults, a low-GI diet reduced muscle triglycerides, a marker of insulin resistance.12 In addition, consuming high levels of high-glycemic carbohydrates causes enhanced appetite and a tendency to overeat.13,14 However, a complete reversal of consuming refined carbohydrates and saturated fats and switching to a complex carbohydrate, protein-rich diet with healthy fatty-acid ratios is not likely to...

Hypericin and hyperforin

In a human study, 13 healthy volunteers were given 300mg standardized extract H. perforatum TID for 14 d. 24 h urinary excretion ratios of 6-betahydroxycortisol cortisol were used as an index of CYP3A4 activity. A significant increase, from zero up to around 2.5x over base was found in urinary ratios in 12 subjects, suggestive of 3A4 induction.

Differential Proteome Profiles during PMIs

Sheath blight disease caused by R. solani can serve in future studies of rice-fungal interactions because little is known about the host response to the pathogen infection at the protein levels. Lee et al. 12 reported the first proteomic analysis combined with a genetic analysis on the rice response to the fungus. 2-DGE followed by SYPRO Ruby staining and MS MS analysis using ESI-QTOF-MS was subjected to detect and identify proteins differentially expressed. Eleven of 14 identified proteins (putative 3-P hydroxysteroid dehydrogenase isomerase protein, stromal APX, putative alpha 1 subunit of 20 S proteasome, RuBisCO activase, RuBisCO large chain, putative chap-eronin 60, j precursor 14-3-3-like protein, endosperm lumenal Bip, two putative chitinase, and GAPDH A subunit) were induced only in the resistant line against R. solani. They can be categorized into the presumed functional groups such as antifungal activity, signal transduction, energy metabolism, photosynthesis, molecular...

Sex hormones and thyroid hormones

And plasma growth hormone are enhanced, and the concentration of thyrotrophin remains unchanged but adrenocorticotrophin is suppressed. Also, the pattern of plasma androgen is altered. The levels of plasma Cortisol and to a lesser extent of plasma aldosterone are increased.

Research findings on psychological effects of lavender

Based on Buchbauer et al.'s (1993) results on the sedative effects of lavender on mice, Bradshaw et al. (1998) found that lavender straw present during transportation halved the incidence of travel sickness symptoms from the 30 per cent measured in pigs bedded with wheat straw, although stress levels, as measured by salivary cortisol, were not changed. They suggest that this could either be due to the sedative effect of lavender or to the distracting effect of the novel odour.

Adaptogenic Botanicals

Licorice (Glycyrrhiza glabra) retains modest glucocorticoid activity via its cortisol-sparing effect. Glycyrrhizin, a component of licorice, inhibits the activity of 11-beta-hydroxy-steroid dehydrogenase, which leads to an increase in cortisol half-life.29 Glycyrrhizin can bind to glucocorticoid and mineralocorticoid receptors, exerting a weak mimetic effect.30 In addition, licorice inhibits phospholipase A2, which then lowers inflammatory prostaglandin and leu-kotriene production. Given to animals exposed to experimental physical stress, ashwagandha produces anti-stress and anabolic activity similar to that of Asian ginseng (Panax ginseng).31 When administered to animals, Siberian ginseng, more accurately known as eleuthero, (Eleutherococcus senticosus) inhibited many of the biologic changes accompanying extreme stress, such as adrenal weight changes, increasing cortisol levels, and blood-sugar levels.32 A large body of research has demonstrated an enhanced response to physical or...

Diagnosing Adrenal Dysfunction

The 24-Hour Salivary Cortisol Pattern This pattern consists of four points 7 am-8 am, noon, 4 pm-5 pm, and 11 pm-midnight. Research suggests that measuring salivary, as opposed to serum, cortisol and DHEA levels may be the best indication of adrenal function.71-74 Yet controversy exists concerning the complete validity of such testing methods because of potentially confounding variables, such as dietary interference, diurnal variations in salivary production and viscosity, oral contaminants, and the potential presence of gingival disease or problems regarding the mouth ecology. A 24-hour urine test may provide a better overall picture of glandular function in contrast to a spot urine or blood test, which provides merely a ''snapshot'' of physiological functioning. In addition, the 24-hour urine measures free cortisone to cortisol ratio, which is useful to identify issues related to 11-beta-hydroxysteroid dehydrogenase, the enzyme that converts active cortisol to inactive cortisone....

Conclusions

A particularly interesting study revealed that cortisol strongly fluctuates with increases and decreases in negative affect. The parameters of this research included testing salivary cortisol levels of 30 healthy young men experiencing an activating and humorous video, a speech stressor, and a resting control. The study researchers found that negative affect increased during the speech but strongly decreased during the video. The researchers concluded that their results suggested that the HPA axis is a dynamic system that is influenced by changes in negative affect independently of the experience of generalized activation.125 An increasing amount of research points to the relation between some emotional states and sympathetic nervous system over-activity.3,126 This research confirms that the mind and body can no longer be looked on as separate moieties. Whatever the underlying mechanism that connects mind and body, the writing on the wall is quite clear medicine can no longer separate...

Vitamin C

Vitamin C increases T-lymphocyte activity, phagocyte function, leukocyte mobility, and interferon production. Studies have shown abnormal phagocytes, antibodies, and cytokines in women with endometriosis, so vitamin C may be therapeutically useful. Because it is an antioxidant, vitamin C can protect cells from reactive oxygen species known to cause tissue damage and disease. This protective effect may also prevent tissue damage from dioxin and PCBs. Women with high estrogen levels, oral contraceptive users, and nicotine users will have increased vitamin C excretion and measurably lower plasma levels, and these women might require higher levels of supplementation.42,43

Case Discussion

Recently, a 57-year-old menopausal patient presented with numerous hormone-related symptoms, including hot flashes, night sweats, irritability, and vaginal dryness, that had not responded to standard hormonal treatment provided by her previous physician. After having the patient complete a 24-hour urine collection for a hormone-profile analysis, her clinical picture became substantially clearer. Her laboratory values reflected low progesterone metabolites, pointing to low progesterone levels. She had low normal testosterone and androgen levels and substantially lower-than-optimal cortisone and cortisol levels. This correlated well with her previous experience, when she had taken progesterone, either orally or transder-mally, and felt better. However, her severe atopic vaginitis with ulcerations strongly correlated with an effect caused by lower-than-normal estrogen. Thus, she had taken a preparation consisting of estradiol, estrone, and estriol. However, she experienced symptoms of...

Etiology

Many studies have indicated that either insulin resistance or endocrine dysfunction can cause the symptoms associated with this condition.2 Many theories have been suggested to explain the primary defect that causes PCOS pathology. They include abnormal insulin action and secretion, endocrine abnormalities causing an increased luteinizing hormone (LH) pulse frequency and amplitude, increased androgen production from the ovaries, and abnormal cortisol metabolism.2 There is also evidence that genetic factors play a role in PCOS.3 Eating disorders, such as bulimia and binge eating, have also been associated with this condition.4

Lignans

Hydroxymatairesinol Lignan

Lignans of Myristica argentea including erythro-austrobailignan-6 and meso-dihydroguaiaretic acid, myristargenol A, and myristargenol B from the aril of the seeds, show some levels of activity against Streptococcus mutans. Erythro-austrobailignan-6, meso-dihydroguaiaretic acid, and nectandrin-B (Fig. 5.185) exert an antiproliferative effect on MCF-7 cells as well as antioxidant activity on the 1,1-diphenyl-2-picrylhydrazyl radical. In addition, Nectandrin-B inhibits the enzymatic activity of 17p hydroxysteroid dehydrogenase and antiaromatase activities (Fig. 5.263).

Liliaceae

Cows (Watt and Breyer-Brandwijk, 1962). C The stems contain 2 of a mixture of five pregnane glycosides. One of two recently reported genins contains a dihydroacetone group like Cortisol but has different stereochemistry at C-14andC-17(Schauber al., 1968 Stockel et al., 1969). P Not only does this plant seem to act via the adrenal-pituitary axis but convulsions similar to those produced by strychnine, which result in paralysis and death, have been observed in cattle (sheep) after they have eaten the plant (Steyn, 1937). Therefore this plant should not be considered for human or veterinary applications

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